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| Hydrosalpinx |
| Etiology •Chlamydia trachomatis, Neisseriae gonorrhea, variety of coliforms and other organisms result in an infection •resolution of the acute infection may result in hydrosalpinx |
| Pathogenesis •fusion of the fimbriated end of the tube following acute inflammation and is •proteolysis of neutrophils and debris leaves serous fluid, |
| Epidemiology •women in the reproductive age group •most are secondary to sexually transmitted disease, namely Chlamydia trachomatis and Neisseriae gonorrhea •others are associated with postpartum, postabortal, post-instrumentation, or with an IUD and can be due to a variety of organisms •part of the syndrome termed pelvic inflammatory disease (P.I.D) |
| General Gross Description •dilated fallopian tube which is filled with clear, colorless, watery fluid •tubal wall is thin •external surface may exhibit adhesions |
| General Microscopic Description •dilated, thin walled tube with flattened epithelium •ciliated and secretory tubal epithelium can be recognized |
| Clinical Correlation •sequel to acute pelvic inflammatory disease •associated with infertility |
| References • Cotran RS, Kumar V, Robbins SL: Robbins Pathologic Basis of Disease. 5th ed. Philadelphia, W.B. Saunders, 1994, pp. 1038-9. |
| Hydrosalpinx |
| Synopsis by: Melinda Sanders M.D. (T86100M33300)[1] |
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