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| Pyosalpinx |
| Etiology •secondary to bacterial infection usually Chlamydia trachomatis or Neisseria Gonorrhae when sexually transmitted or •staphylococci, streptoccoci, or coliforms •organisms reach fallopian tube by ascending through cervix and across endometrium |
| Pathogenesis •suppurative acute inflammatory response to organisms within the fallopian tube •may occur rapidly following vaginal inoculation with organism (3-5 days in experimental models) •may be facilitated by estrogen •fusion of the end of the tube results in purulent accumulation within the dilated tube, |
| Epidemiology •part of Pelvic Inflammatory Disease (P.I.D.) which may be •sexually transmitted disease or •follow delivery, abortion, instrumentation, or IUD placement •disease of reproductive age women |
| General Gross Description •enlarged fallopian tube with hyperemic external surface which may show serositis •lumen filled with pus |
| General Microscopic Description •acute inflammatory infiltrate within the lumen and involving epithelium of the tube |
| Clinical Correlation •uterine tenderness and pain accompanying vaginal discharge •systemic symptoms of fever and malaise may be accompanied by leukocytosis •treatment with antibiotics •risk of infertility pronounced with development of pyosalpinx |
| References • Cotran RS, Kumar V, Robbins SL: Robbins Pathologic Basis of Disease. 5th ed. Philadelphia, W.B. Saunders, 1994, pp. 1039 |
| Pyosalpinx |
| Synopsis by: Melinda Sanders M.D. (T86100M40460)[2] |
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