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| Bronchopneumonia |
| Etiology • Variety of aspirated organisms. • Organism dependent on whether community acquired in previously healthy patient (more likely Streptococcus) or • Community acquired in patient with depressed pulmonary defenses such as a patient with chronic bronchitis (more likely Klebsiella or Pseudomonas spps) or • Hospital acquired |
| Pathogenesis • Aspiration of organisms results in inflammation and necrosis of underlying parenchyma • Tends to scar if alveolar septae are destroyed, |
| Epidemiology • Common in hospitalized patients and contributes to the cause of death in moribund patients • Most common community acquired pneumonia as well |
| General Gross Description • Patchy distribution particularly around small airways • Nodular, elevated, firm, airless regions • Range from red to gray depending on age of the lesion • Can become confluent to mimic lobar pneumonia |
| General Microscopic Description • Bronchocentric lesions • Neutrophils fill airway and surrounding alveoli • Parenchymal destruction depends on organism • Uninvolved parenchyma may contain acellular pink edema |
| Clinical Correlation • Clinical course dependent on underlying disease processes • Patients present with fever, cough and purulent sputum |
| References • Cotran RS, Kumar V, Robbins SL: Robbins Pathologic Basis of Disease. 5th edition. Philadelphia, W.B. Saunders, 1994, pp. 694-698. |
| Bronchopneumonia |
| Synopsis by: Melinda Sanders M.D. (T28000M40000)[122] |
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